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IAEDP Core Course 1 · Study companion

The whole curriculum, made easier to understand.

Move from a plain-language explanation to the verbatim slide, speaker notes, and original visual—without losing the source.

Explore the course map
Biology
Psychology
Social world
81source-traceable slides
81slide lessons
8learning modules
25original media files
100%verbatim source access

A four-layer learning loop

Understand it. Master it. Remember it. Verify it.

  1. 1Learn the plain-language idea

    A careful explanation gives you a mental foothold.

  2. 2Master the testable details

    Every slide names its takeaways, definitions, lists, numbers, and distinctions.

  3. 3Attach a memory hook

    A short phrase makes the larger idea easier to retrieve.

  4. 4Return to the exact source

    Every lesson keeps the verbatim curriculum one click away.

Complete learning path

Eight modules. One connected picture.

The order follows the original 81-slide deck, from foundational models through diagnosis, diversity, safety, evaluation, teamwork, and application.

Slides 126 · 26 lessons

Foundations and contributing factors

Start with the course goals, then connect biological, psychological, and social factors.

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Slides 2745 · 19 lessons

Diagnoses and clinical distinctions

Study the criteria, severity language, remission terms, and differences among feeding and eating disorders.

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Slides 4663 · 18 lessons

Diversity, stereotypes, and access

See how identity, culture, age, disability, resources, and access affect detection and care.

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Slides 6467 · 4 lessons

Suicide and safety assessment

Review the course’s suicide-risk framing, assessment prompts, and treatment models.

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Slides 6872 · 5 lessons

Evaluation and levels of care

Organize the information an evaluation needs and compare treatment settings by support intensity.

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Slides 7375 · 3 lessons

Multidisciplinary treatment

Understand why eating-disorder care is collaborative and what each role contributes.

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Slides 7679 · 4 lessons

Case study: Carmela

Apply the course to a developing case without jumping prematurely to one conclusion.

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Slides 8081 · 2 lessons

Course review and references

Close the course, revisit the learning path, and keep the original reference layer available.

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Preview of the original Core Course 1 PowerPointOriginal slide visual

Source stays attached

Simplified never means separated from the curriculum.

Every lesson includes the extracted editable text, the speaker notes when present, the full slide render, and links to embedded image files. The teaching layer helps you understand; the source layer lets you verify.

Course-wide study guide

Things to remember after all 81 slides.

This is the long-form review layer: eight connected ideas, the questions that keep them clinically careful, and a final set of one-line recall cues.

01

Slides 4–7, 24–26

Begin with the whole picture

An eating disorder is not explained by one cause. Biology, psychology, relationships, culture, resources, and life events can interact.

  • Predisposing factors prepare the ground; precipitating factors can act like a spark; perpetuating factors can keep the cycle going.
  • The factor that helped start the illness may be different from the factor maintaining it now.
  • A theory is a useful lens for asking questions—not proof that one explanation fits every person.
  • The bio-psycho-social model protects against simplistic blame because it requires attention to body, mind, and world.
Ask yourselfCan I name biological, psychological, interpersonal, and sociocultural questions I would still need to ask?
02

Slides 8–14, 26

Biology changes vulnerability, not destiny

Genes, neurobiology, nutrition, energy balance, and the gut may influence risk and illness—but none acts as a personal prediction by itself.

  • Heritability is a population statistic. It does not mean an individual is a certain percentage genetically destined to develop an illness.
  • Malnutrition is not only a consequence; it can worsen cognition, mood, distress, and physical instability, helping maintain the cycle.
  • Group-level brain findings do not diagnose one person.
  • Gut-microbiota research is developing and should be understood as one possible layer within a much larger system.
  • Weight cycling and the difficulty of long-term weight-loss maintenance belong in the clinical context.
Ask yourselfAm I translating a research finding into an individual prediction that the research does not support?
03

Slides 27–45

Diagnoses describe patterns, not people

Diagnostic criteria organize recurring symptoms, duration, distress, impairment, and exclusions. They do not measure a person’s worth or how much care they deserve.

  • Anorexia nervosa centers on restriction leading to significantly low weight, fear or interference with weight gain, and disturbance in body experience or recognition.
  • Bulimia nervosa includes recurrent binge eating with loss of control and recurrent compensatory behavior at the required frequency and duration.
  • Binge-eating disorder includes recurrent binges and distress without the recurrent compensation characteristic of bulimia nervosa.
  • ARFID involves avoidance or restriction with meaningful consequences, without the AN/BN body-shape pattern.
  • Rumination disorder, pica, OSFED, and UFED each have their own role; clinically important symptoms do not become unimportant because they do not fit a stereotype.
  • Severity and remission describe the current clinical picture. They are not moral grades.
Ask yourselfWhat required pattern, duration, impairment, and exclusion distinguishes the diagnosis I am considering?
04

Slides 48–62

Recognition must be wider than the stereotype

Eating disorders affect people across bodies, ages, genders, races, cultures, abilities, income levels, and locations—while recognition and access remain unequal.

  • A narrow mental image of who has an eating disorder creates a narrow detection system.
  • Minority stress can include both external experiences of prejudice and the internal burden of anticipating or absorbing it.
  • Food insecurity and low socioeconomic status can increase vulnerability while making specialized treatment harder to reach.
  • Disability, mid-life and older age, and male identity are not protective against eating disorders.
  • Transgender, gender-diverse, racialized, and LGBTQ+ communities can face intersecting stress, bias, and access barriers.
  • Global prevalence does not mean every culture expresses or explains symptoms in the same way.
Ask yourselfWhose symptoms could I miss if I only look for the most familiar presentation?
05

Slides 20–22, 63

Weight stigma is part of the clinical environment

Weight stigma can come from many people and systems, affect people across weight categories, and change whether someone seeks or receives appropriate care.

  • Bias is the belief or attitude; discrimination is the harmful behavior or unequal treatment.
  • Examples can include taunts, physical abuse, microaggressions, stereotypes, images, and language.
  • Potential perpetrators include media, peers, employers, teachers, strangers, friends, family, and health professionals.
  • Effects can be emotional, social, physical, and behavioral—including avoidance of medical care.
  • Equality gives everyone the same support; equity responds to different barriers so people can reach a fair opportunity.
Ask yourselfIs a weight assumption preventing me from considering another medical, psychological, or social explanation?
06

Slides 64–67

Safety assessment must be direct and whole-person

Eating disorders can carry both medical and suicide risk. Neither form of safety can be assumed from appearance, diagnosis, or one reassuring answer.

  • Ask directly about suicidal thoughts, timing, plans or methods, immediacy, past behavior, and consequences.
  • Take every form of self-harm seriously.
  • Different suicide models highlight belonging, burdensomeness, psychological pain, acquired capacity, and escape from self-awareness.
  • A study guide cannot replace qualified clinical judgment, local emergency procedures, supervision, or an individualized safety plan.
Ask yourselfHave I asked clearly enough to understand immediacy, intent, means, prior behavior, and protective context?
07

Slides 68–72

Match the level of care to the full clinical picture

Levels of care differ in structure, monitoring, medical capability, meal support, treatment hours, and independence.

  • A thorough evaluation gathers medical, psychiatric, behavioral, developmental, family, social, and treatment-readiness information.
  • Inpatient settings provide 24-hour operation and frequent monitoring, but specialized medical and psychiatric programs differ in their available interventions.
  • Residential care is 24/7; partial hospital and intensive outpatient care provide scheduled daily or weekly structure; outpatient care relies on greater independence.
  • Meal supervision and on-site medical support generally change as care becomes less intensive.
  • The right placement is a clinical matching decision, not a simple label or a measure of how sick someone looks.
Ask yourselfWhat medical, psychiatric, behavioral, support, and functional needs must this setting safely meet?
08

Slides 73–80

Coordinate the team, then apply the whole framework

Eating-disorder treatment crosses medical, nutritional, psychological, functional, family, and social systems, so coordinated care matters.

  • Each discipline contributes a different part of the picture; communication is what turns separate expertise into a team.
  • The client is an active team member whose goals, experience, and communication guide decision-making.
  • Families, friends, schools, and other supports may hold useful collateral information when appropriate.
  • In Carmela’s case, insulin manipulation, diabetes, bullying, school context, development, motivation, timing, and the health event all matter.
  • Gather the whole story before naming the pattern, then identify maintaining forces and the team needed to respond.
Ask yourselfWhat information is missing, who may hold it, and which disciplines need to coordinate next?

If you remember only twelve things

A final quick-scan before you close the course.

  1. 01No single cause tells the whole story.
  2. 02Ground, spark, wind: predispose, precipitate, perpetuate.
  3. 03The cause of onset may not be the cause of maintenance.
  4. 04Heritability is not destiny.
  5. 05Malnutrition can become fuel for the cycle.
  6. 06A diagnosis describes a pattern, not a person.
  7. 07There is no single eating-disorder face.
  8. 08Bias narrows what clinicians notice.
  9. 09Ask about suicide directly and follow qualified safety procedures.
  10. 10Level of care should match medical, psychiatric, and functional need.
  11. 11Many roles contribute; coordination creates the whole picture.
  12. 12Keep every simplified explanation traceable to its source.